PL EN


Preferences help
enabled [disable] Abstract
Number of results
Journal
2013 | 8 | 3 | 354-357
Article title

Urine saturation and promoter/inhibitor parameters and ratios in renal stone disease caused by ceftriaxone

Content
Title variants
Languages of publication
EN
Abstracts
EN
During ceftriaxone treatment of subdural empyema caused by Streptococcus intermedius urinary and biliary stones were noticed. Increased levels of urinary calcium excretion were detected during ongoing treatment in comparison with 2 months check-up. There were no significant changes in the promoter/inhibitor urolithiasis parameters, oxalate, citrate, urate, cistine, glycosaminoglycans or their ratios. Urine saturation was calculated using EQUIL 2 computer programme (calcium oxalate, brushite) and it was normal. Probable trigger for the ceftriaxone/calcium hydroxy carbonate phosphate mixture of stones was a critical boost of solubility products caused by ceftriaxone treatment and phospnate urine content with a subsequent large-scale spontaneous precipitation of crystals.
Publisher

Journal
Year
Volume
8
Issue
3
Pages
354-357
Physical description
Dates
published
1 - 6 - 2013
online
17 - 4 - 2013
Contributors
  • Department for Pediatric Nephrology, University Children’s Hospital Zagreb, Zagreb School of Medicine, Kišpatićeva 12, 10000, Zagreb, Croatia, danko.milosevic@zg.t-com.hr
author
  • Department for Pediatric Infectious diseases, School of Medicine, Zagreb University, Zagreb, Croatia
author
  • Department for Physical chemistry, Institute „Ruđer Bošković“, Bijenička cesta 54, 10000, Zagreb, Croatia
  • Department of Anesthesiology and Intesive Care, University Clinical Centre Zagreb, Zagreb, Croatia
  • Department for Pediatric Nephrology, University Children’s Hospital Zagreb, Zagreb School of Medicine, Kišpatićeva 12, 10000, Zagreb, Croatia
author
  • Department for Pediatric Nephrology, University Children’s Hospital Zagreb, Zagreb School of Medicine, Kišpatićeva 12, 10000, Zagreb, Croatia
  • Department for Pediatric Nephrology, University Children’s Hospital Zagreb, Zagreb School of Medicine, Kišpatićeva 12, 10000, Zagreb, Croatia
References
  • [1] Schaad UB., Wedgwood-Krucko J., Tschaeppler H. Reversible ceftriaxone-associated biliary pseudolithiasis in children. Lancet, 1988, 8625, 1411–1413 http://dx.doi.org/10.1016/S0140-6736(88)90596-X[Crossref]
  • [2] de Moor RA., Egberts AC., Schröder CH. Ceftriaxone associated nephrolythiasis and biliary pseudolithiasis. EUR J Pediatr, 1999, 158, 975–977 http://dx.doi.org/10.1007/s004310051261[Crossref]
  • [3] Bor O., Dinleyici EC., Kebapci M., Aydogdu SD. Ceftriaxone-associated biliary sludge and pseudocholelithiasis during childhood: a prospective study. Pediatr Int, 2004, 46, 322–324 http://dx.doi.org/10.1111/j.1328-0867.2004.01884.x[Crossref]
  • [4] Acun C., Erdem LO., Söğüt A., Erdem CZ., Tomaç N., Gündoğdu S., et al. Gallbladder and urinary tract precipitations associated with ceftriaxone therapy in children: a prospective study., Ann Trop Pediatr, 2004, 24, 25–31 http://dx.doi.org/10.1179/027249304225013349[Crossref]
  • [5] Gargollo PC., Barnewolt CE., Diamond DA. Pediatric ceftriaxone nephrolythiasis. J Urol, 2005, 173, 577–578 http://dx.doi.org/10.1097/01.ju.0000148801.12464.b3[Crossref]
  • [6] Mohkam M., Karimi A., Gharib A., Daneshmand H., Khatami A., Ghojevand N., et al. Ceftriaxone associated nephrolythiasis: a prospective study in 284 children. Pediatr nephrol, 2007, 22, 690–694 http://dx.doi.org/10.1007/s00467-006-0401-2[WoS]
  • [7] Avci Z., Koktener A., Uras N., Catal F., Karadag A., Tekin O., et al. Nephrolythiasis associated with ceftriaxone therapy: a prospective study in 51 children. Arch Dis Child, 2004, 89, 1069–1072 http://dx.doi.org/10.1136/adc.2003.044156[Crossref]
  • [8] Karlizcek SB., Döring S., Vogt S., et al.Ceftriaxone-associated nephrolythiais. Two case reports. Monatsschr Kinderheilkd, 1996, 144, 702–706
  • [9] Kimata T., Kaneko K., Takahashi M., Hirabayashi M., Shimo T., Kino M. Increased urinary calcium excretion caused by ceftriaxone: possible association with urolythiasis. Pediatr nephrol, 2012, 27, 605–609 http://dx.doi.org/10.1007/s00467-011-2038-z[Crossref][WoS]
  • [10] Lozanovski VJ., Gucev Z., Avramoski VJ., Kirovski I., Makreski P., Tasic V. Ceftriaxone associated urolithisis in a child with hypercalciuria. Hippokratia, 2011, Apr–Jun 15(2),181–183
  • [11] Milošević D., Batinić D. Blau N., Konjevoda P., Štambuk N., Votava-Raić A., et al.Determination of urine saturation with computer program Equil 2 as a method for estimation of the risk of urolithiasis. J Chem Inf Comput Sci, 1998, 38(4), 646–650 http://dx.doi.org/10.1021/ci9701087[Crossref]
  • [12] Batinić D., Milošević D., Blau N., Konjevoda P., Štambuk N., Barbarić V., et al. of the stone promoters/inhibitors ratios in the estimation of the risk of urolithiasis. J Chem Comput Sci, 2000, 40(3), 607–610 [Crossref]
  • [13] Werness P., Brown CM., Smith LH., Finlayson B., EQUIL II: a BASIC computer program for the calculation of urinary saturation. J Urol, 1985, 134, 1242–1244
  • [14] Witten IH., Frank E. Data mining:practical machine learning tools and techniques with Java implementations. Morgan Kaufman: San Francisco, 2000.
  • [15] Milošević D., Batinić D., Konjevoda P., Blau N., Štambuk N., Nižić L.j, et al. of calcium, oxalate, and citrate interaction in idiopathic calcium urolithiasis in children. J Chem Inf Comput Sci, 2003, 43(6), 1844–1847 http://dx.doi.org/10.1021/ci020060j[Crossref]
  • [16] Chutipongtanate S. and Thongboonkerd V. Ceftriaxone crystallization and it potential role in kidney stone formation. Biochem Biophys Res Commun., 2011 Mar, 406(3), 396–402 http://dx.doi.org/10.1016/j.bbrc.2011.02.053[Crossref]
  • [17] Reid DG., Jackson GJ., Duer MJ., Rodgers AL. Apatite in kidney stones is a molecular composite with glycosaminogycans and proteins: evidence from nuclear magnetic resonance spectroscopy, and relevance to Randall’s plaque, pathogenesis and prophylaxis. J Urol, 2011 Feb, 185(2), 725–730 http://dx.doi.org/10.1016/j.juro.2010.09.075[WoS][Crossref]
  • [18] Hesse A. and Heimbach D. Causes of phosphate stone formation and the importance of metaphylaxis by urinary acidification: a review. World J Urol, 1999 Oct, 17(5), 308–315 http://dx.doi.org/10.1007/s003450050152[Crossref]
  • [19] Battino BS., DeFOOR W., Coe F., Tackett L., Erhard M., Wacksman J., Sheldon CA., Minevich E. Metabolic evaluation of children with urolithiasis: arer adult references for supersaturation appropriate?. J Urol, 2002 Dec, 168(6), 2568–2571 http://dx.doi.org/10.1016/S0022-5347(05)64217-6[Crossref]
  • [20] Patzer L., van’t Hoff W., Shah V., Halson P., Kasidas GP., Colin S., de Bruyn R., Baratt TM., Dillon MJ. Urinary supersaturation of calcium oxalate and phosphate in patients with X-linked hypophosphatemic rickets and in healthy school children. J Pediatr, 1999 Nov, 135(5), 611–617 http://dx.doi.org/10.1016/S0022-3476(99)70060-0[Crossref]
  • [21] Shiffman M., Keith FB., Moore EW. Pathogenesis of seftriaxone-associated biliary sludge. In vitro studies of calcium-ceftriaxone binding and solubility. Gastroenterology, 1990 Dec, 99(6), 1772–1776
  • [22] Malsy A. and Bohner M. Brushite conversion into apatite. European Cells and Minerals Vol, 10. Suppl. 1, 2005, (page 28)
  • [23] Asplin J., Parks J., Lingeman J., Kahnoski R., Mardis H., Lacey S., Goldfarb D., Grasso M., Coe F. Supersaturation and stone composition in a network of dispersed treatment sites. J Urol, 1998 Jun, 159(6), 1821–1825 http://dx.doi.org/10.1016/S0022-5347(01)63164-1[Crossref]
Document Type
Publication order reference
Identifiers
YADDA identifier
bwmeta1.element.-psjd-doi-10_2478_s11536-012-0091-z
JavaScript is turned off in your web browser. Turn it on to take full advantage of this site, then refresh the page.