Full-text resources of PSJD and other databases are now available in the new Library of Science.
Visit https://bibliotekanauki.pl
Preferences help
enabled [disable] Abstract
Number of results

Results found: 3

Number of results on page
first rewind previous Page / 1 next fast forward last

Search results

Search:
in the keywords:  myoglobin
help Sort By:

help Limit search:
first rewind previous Page / 1 next fast forward last
EN
Optimization of training and minimization of injuries are topical for the physical performance of military personnel. Physical and psycho-emotional load, fatigue, sleep deprivation, and dietary limits can lead to the development of oxidative stress (OS) and injuries in specific military training. This study investigated markers of OS and muscle damage in military cadets after a 10-day-long intensive training course and a one-month-long recovery. The sample included 42 cadets (2 females and 40 males) aged from 22 till 34. Myoglobin, catalase activity (CAT), superoxide dismutase activity (SOD), and total antioxidants capacity (TAC) in plasma were measured. OS was assessed by the glutathione index. The results revealed an increasing level of myoglobin, increasing glutathione index, and no changes in CAT, SOD, and TAC during the intensive training course. After the one-month-long recovery, myoglobin was back to normal, the activity of CAT and TAC was higher than before and after the training course, while SOD did not change after the recovery. The glutathione index decreased after the one-month-long recovery, but it was not reached the initial level before the intensive training. In sum, the observed grade of OS positively affected the capacity of the antioxidative system with some sign of a need for a longer rest.
EN
The article presents the case of a 66-year-old man with rhabdomyolysis due to acute alcohol intoxication, manifested clinically by acute kidney injury and a sharp increase in creatine phosphokinase. During hospitalization, the patient suffered from cardiac and respiratory arrest with subsequent death. The post-mortem histopathological examination showed pathologies of numerous organs: putrefactive necrosis, pulmonary edema, pulmonary emphysema, megalocardia, acute myocardial infarction, hemorrhagic erosions of the gastric mucous membrane, atrophy of renal glomerules, colliquative necrosis of the liver, Balser necrosis of the pancreas, ascites, hydrothorax, lacunae in the occipitotemporal region and hydrocephalus. The described case presents a range of clinical problems appearing in the management of patients with severe form of alcohol-induced rhabdomyolysis.
PL
W pracy zaprezentowano przypadek 66-letniego mężczyzny, u którego w wyniku ostrego zatrucia alkoholem doszło do rabdomiolizy manifestującej się klinicznie ostrym uszkodzeniem nerek oraz gwałtownym wzrostem aktywności fosfokinazy kreatynowej. W trakcie hospitalizacji u pacjenta doszło do zatrzymania akcji serca i oddechu, a następnie do zgonu. Posekcyjne badanie histopatologiczne wykazało patologię licznych narządów: proces gnilny, obrzęk płuc, rozedmę płuc, powiększenie serca, świeży zawał serca, nadżerki krwotoczne błony śluzowej żołądka, zanik kłębuszków nerkowych, martwicę rozpływną wątroby, martwicę Balserowską trzustki, wodobrzusze, obecność płynu w jamie opłucnowej, jamy poudarowe okolicy skroniowo-potylicznej oraz wodogłowie. Opisany przypadek prezentuje zakres problemów klinicznych występujących w postępowaniu z pacjentem z ciężką postacią rabdomiolizy indukowanej spożyciem alkoholu.
first rewind previous Page / 1 next fast forward last
JavaScript is turned off in your web browser. Turn it on to take full advantage of this site, then refresh the page.