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Predictive factors of complications post intracranial meningioma surgery leading to early unplanned reoperations — a single center study

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Kwinta B. M., Krzyżewski R. M., Kliś K. M., Dragan M. M., Donicz P., Gackowska M., Stachura K., Moskała M.
Folia Medica Cracoviensia
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2018
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vol. 58
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issue 2
2
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Meningoma located primarily within the internal auditory canal - a case report

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Mogla M., Laskus A., Karchier E., Niemczyk K.
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vol. 6(1)
8-11
EN
A 34-year old male presented with a 5-year history of hearing loss and tinnitus. MRI revealed a intracanalicular tumor deep inside the internal auditory canal invading partially cochlear ganglion. The mass was surgically resected by middle fossa approach. The histological findings showed meningoma primarily located in IAC. This study presents epidemiology, features and treatment of intracanalicular meningomas.
3
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Meningioma mimicking vestibular schwannoma – a case report

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Wojciechowski T., Drożdż A., Niemczyk K.
Polski Przegląd Otorynolaryngologiczny
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2019
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vol. 8(1)
49-52
EN
Meningiomas are one of the most common intracranial neoplasms, usually located in the supratentorial region. Their location within the posterior cranial fossa is rare, but even in such cases, the diagnosis is possible owing to the characteristic imaging features. In this article, we present a case report of a patient with a meningioma of an internal acoustical meatus resembling vestibulo-cochlear nerve schwannoma in its typical location, causing symptoms such as asymmetric hearing loss, tinnitus and dizziness.
4
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Endoscopic transnasal resection of tuberculum sellae meningioma – case report

88%
Kozok-Paździor M., Dobosz P., Sobolewska A., Sobański D.
Polski Przegląd Otorynolaryngologiczny
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2019
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vol. 8(4)
24-29
EN
Introduction: Tuberculum sellae meningiomas (TSM) represent 5-10% of all intracranial meningiomas. Tumours are located on tuberculum sellae or chiasmatic sulcus of the sphenoid bone. These suprasellar lesions often displace the optic nerves causing visual impairment that is commonly the presenting symptom. Tuberculum sellae meningiomas are traditionally operated by transcranial approach. There is an alternative method to remove tumours of anterior skull base by using endoscopic transnasal surgery. Methods: In this article we describe a case of the patient with tuberculum sellae meningioma. Results: 63-year old women was diagnosed due to a headache, facial sensation defects on the right and peripheral vision loss. Ophthalmology examination showed bitemporal vision loss up to 10-15%. Using magnetic resonance imaging scan the tumour of diameter 8.1x8.0mm was located in tuberculum sellae. The endoscopic transsphenoidal operation was made confirming diagnosis of meningioma. In postoperative period the symptoms was almost completely minimalized. Conclusion: The treatment of choice for tuberculum sellae meningiomas is a surgery. Neurosurgeons have two options to remove the tumour: by transcranial approaches or via endoscopic transsphenoidal surgery with rhinologist. The decision of the optimal surgical technique should be individually made. Knowing possible complications during endoscopic approach, the benefits prevail. Cerebrospinal fluid leak is still challenging but using nasoseptal flap (Hadad’s flap) makes reconstruction easier. Recovery is quicker and postoperative results are promising.
5
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Badania kliniczno-molekularne w oponiakach

63%
Jaskólski D. J.
Aktualności Neurologiczne
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2012
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vol. 12
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issue 2
74-103
EN
Aim: The aim of our study was to evaluate the frequency of deletions on chromosomes 1, 9, 10, 14, 18 and 22 in 75 benign and 15 atypical meningiomas and correlate them with clinical findings. Methods: Paired normal and tumour DNA samples obtained from the patients operated on, were analyzed for loss of heterozygosity (LOH), using 24 microsatellite markers and PCR techniques. Results: Statistical analysis showed that deletions on chromosomes 14 and 18 were significantly associated with WHO grade of the meningiomas (p=0.048 and p=0.03, respectively). In addition, LOH on chromosome 14 was significantly associated with tumour size (p=0.048), as the risk of developing a tumour larger than 4 cm in diameter was 6-times greater than the risk of developing tumour with diameter below 4 cm. The most frequent genetic abnormality inmeningiomas is 22 LOH, which was confirmed in the present study in which high frequency of such abnormality was observed (66%). There was a clear associations between chromosome 22 status and histological subtype. LOH on chromosome 22 was more frequent in fibrous meningiomas than in the meningothelial variant (p=0.001). Besides that, there was a relationship between 22 LOH status and tumour location: the frequency of LOH in skull base meningiomas was significantly lower compared to parasagittal meningiomas (p=0.0004). Conclusions: These results indicated that allelic loss on chromosomes 9, 10, 14, 18 and 22 may be associated with meningioma pathogenesis and progression.
PL
Cel: Celem pracy była ocena częstości występowania delecji na chromosomach 1., 9., 10., 14., 18. i 22. w 75 łagodnych i 15 atypowych oponiakach oraz skorelowania ich z niektórymi danymi klinicznymi. Materiał i metoda: Sparowane próbki prawidłowego, pochodzącego z leukocytów krwi obwodowej DNA oraz DNA oponiaka zostały zbadane pod kątem utraty heterozygotyczności (LOH) za pomocą 24 markerów mikrosatelitarnych i techniki reakcji łańcuchowej polimerazy (PCR). Materiał pochodził od chorych operowanych. Wyniki: Analiza statystyczna wykazała, że delecje na chromosomach 14. i 18. były w istotny sposób związane ze stopniem WHO oponiaków (odpowiednio p=0,048 i p=0,03). Co więcej, LOH na chromosomie 14. był istotnie statystycznie związany z rozmiarem oponiaka (p=0,048), jako że ryzyko wzrostu nowotworu o średnicy powyżej 4 cm było 6-krotnie większe niż ryzyko rozwoju mniejszego guza. Najczęściej występującą nieprawidłowością genetyczną w oponiakach jest LOH na chromosomie 22., co zostało potwierdzone w przedstawionym materiale, w którym zmianę tę zaobserwowano w 66% przypadków. Występował silny związek pomiędzy zmianami na chromosomie 22. a podtypem histologicznym nowotworu. Utrata heterozygotyczności na chromosomie 22. była częstsza w oponiakach włóknistych niż w meningotelialnych (p=0,001). Ponadto obserwowano związek pomiędzy obecnością LOH na 22. chromosomie a umiejscowieniem oponiaka: częstość LOH w guzach podstawy czaszki była znacząco mniejsza w porównaniu z oponiakami przystrzałkowymi (p=0,0004). Wnioski: Powyższe wyniki wskazują, że utrata heterozygotyczności na chromosomach 9., 10., 14., 18. i 22. może mieć związek z patogenezą i progresją oponiaka.
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