Full-text resources of PSJD and other databases are now available in the new Library of Science.
Visit https://bibliotekanauki.pl
Preferences help
enabled [disable] Abstract
Number of results

Results found: 3

Number of results on page
first rewind previous Page / 1 next fast forward last

Search results

help Sort By:

help Limit search:
first rewind previous Page / 1 next fast forward last
EN
Aims and scope: Delayed Onset Muscle Soreness (DOMS) develops after intense physical activity and its mechanisms are due to inflammation. Kinesiology Taping (KT) improves microcirculation, supports myofascial functions and relieves the tissue. The aim of this study was to verify whether KT has an analgesic action in the DOMS and whether somatotype is associated with this action. Materials and Methods: There were 20 healthy subjects aged 27.7 ±6.4 years with moderate or high physical activity included into the study. The training with emphasis on eccentric muscle work was performed. Somatotype of respondents was assessed by Heath-Carter method. While DOMS occurred, KT muscle application on one of the limbs was done. For the next five days subjects filled out questionnaires in which they served intensity of pain on the basis of Visual Analogue Scale (VAS). Results and conclusions: In the limb where KT application was used a significant (p < 0.05) reduction in the intensity of DOMS compared to the limb without application was observed. It was found that the somatotype has no effect on the reduction of DOMS (p > 0.05). Conclusions: KT exhibits analgesic properties in DOMS. Somatotype has no relation to the effectiveness of KT analgesic efficacy in DOMS.
EN
Neuropsychiatric disorders are one of the most common problems in the modern world. In the European Union alone, at least 164 million cases of mental problems were reported in 2016. Despite many years of investigations and enormous resources devoted to scientific research, the causes of some of these diseases have not been clearly identified, and, in the case of others, treatment options are significantly limited. Although the brain weight accounts only for about 2 percent of the body weight, its oxygen consumption accounts for up to 20% of the resting energy metabolism. As opposed to other organs, the brain does not accumulate energy; therefore its functioning depends on the continuous supply of the main energetic substrate, i.e. glucose. At the same time, the physiology of every cell in the human body depends on the availability of regulatory components, including vitamins and trace elements, which are derived from the diet. As evidenced by the basics of cell biology, certain nutrients are essential for proper neuronal functioning and, consequently, nerve signal transmission. Nutrients are also known to be metabolically utilised by intestinal microorganisms, and the neuroactive and immunocompetent products of these transformations pass through the gut–brain axis into the brain and exert their effects on the structure and function of regions responsible for emotions as well as motor and cognitive abilities. The first part of the article presents information regarding intestinal microbiota as a structural and functional nervous system modulator. Particular attention is paid to intestinal dysbiosis in neurodevelopmental and neurodegenerative disorders, which is currently considered to be the cause and/or a factor determining their clinical course. It was also suggested that supporting gut microbiota with probiotic therapy may be an element of nervous system disease prevention and support the primary treatment of these conditions.
PL
Schorzenia neuropsychiatryczne są jednym z najczęstszych problemów współczesnego świata. W samych tylko krajach Unii Europejskiej w 2016 roku zanotowano przynajmniej 164 mln osób cierpiących na te schorzenia. Mimo wieloletnich dociekań i ogromnych środków przeznaczanych na badania naukowe nie udało się jednoznacznie ustalić przyczyn niektórych spośród tych chorób, a w przypadku innych opcje terapeutyczne są mocno ograniczone. Mózgowie stanowi jedynie blisko 2% masy ciała człowieka, lecz wykorzystuje aż do 20% spoczynkowej przemiany materii. W przeciwieństwie do innych organów mózg nie ma możliwości akumulacji energii, więc jego funkcje uzależnione są od stałego dopływu głównego substratu energetycznego – glukozy. Jednocześnie fizjologia każdej komórki ludzkiego organizmu zależy od dostępności składników regulacyjnych, w tym witamin i pierwiastków śladowych pozyskiwanych z diety. Podstawy biologii komórki dowodzą, iż wybrane składniki żywności są niezbędne do zachowania właściwej funkcji neuronów, a przez to – transmisji sygnałów nerwowych. Wiadomo również, że składniki pokarmowe są wykorzystywane metabolicznie przez zespół mikroorganizmów jelitowych, a neuroaktywne i immunokompetentne produkty tych przemian drogą osi mózgowo-jelitowej przedostają się do mózgowia oraz wpływają na strukturę i funkcje regionów odpowiedzialnych za emocje, zdolności ruchowe i poznawcze. W pierwszej części artykułu przedstawiono wiadomości dotyczące mikrobioty jelitowej jako modulatora struktury i funkcji układu nerwowego. Szczególną uwagę zwrócono na dysbiozę jelitową w  przebiegu zaburzeń neurorozwojowych i neurodegeneracyjnych, rozpatrywaną aktualnie jako przyczyna i/lub czynnik warunkujący ich przebieg kliniczny. Zasugerowano także, że wspieranie mikrobioty jelitowej probiotykoterapią może być elementem profilaktyki chorób układu nerwowego i wspomagania ich leczenia podstawowego.
EN
Background. During the last few decades, adiposity has become a relatively common phenomenon worldwide. The available data on the effects of pro-inflammatory factors in both depression and adiposity has been attracting great attention. Aim. We sought to assess the prevalence of -889C>T IL-1α, -31T>C and -511C>T IL-1β, -330T>G IL-2 and -174G>C IL-6 genes and their association with adiposity and depression in Polish subjects. Methods. A cohort study was conducted in 2013/2014, covering a sample of 297 individuals (217 female and 80 male). Anthropometric data was handled using the BIA analysis method, while for genotyping PCR-RFLP techniques were used. Results. A positive correlation between depression and anthropometric parameters: adipose tissue (in kg) and adipose tissue (in %) (R=0.135 and p=0.02, R=0.114 and p<0.05, respectively) was found. No association between studied polymorphisms and depression was observed. Conclusion. Although it was not possible to demonstrate any influence of the studied polymorphisms as the genetic modulator of depression, authors believe that the presented data are noticeable and may provide the basis for future studies on larger groups.
first rewind previous Page / 1 next fast forward last
JavaScript is turned off in your web browser. Turn it on to take full advantage of this site, then refresh the page.