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The aim of this paper is to present the case of a 70-year-old women with endolymphatic sac tumor and temporal bone destruction treated at Otolaryngology Department of MUG. The patient was admitted to our Department due to a 3-year history of hearing loss, dizziness and ear pain. The first diagnosis was temporal bone tumor connected with von Hippel-Lindau syndrome (VHL). The patient was surgically treated. During intraoperative examination, a neoplasm was determined. The tumor was excised via transmastoid approach with sigmoid sinus skeletonization. After treatment, her pains disappeared. Histopathological and immunohistochemical examination revealed endolymphatic sac tumor. Follow-up CT showed no tumor remission.
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This article presents a case of 42-year-old woman with the iatrogenic transtemporal meningoencephalic herniation. The patient was treated surgically because of chronic otitis media without an expected improvement. Despite new signs and symptoms that appeared after the surgery, no radiological assessment was performed and for another years a wrong treatment was conducted. The authors present diagnostic problems and surgical treatment of meningoencephalic herniation of the temporal bone.
EN
Facial nerve is the main motor supply to the part of facial skeleton system responsible for expressions. The reported rate of iatrogenic injury to the facial nerve in primary mastoid surgeries was 0.6% to 3.7%. Temporal bone is one of the most complex anatomical parts of human body. A variety of facial nerve courses has been described in literature. Normally, horizontal segment of the facial nerve traverses from geniculate ganglion to second genu which is usually situated medial and inferior to lateral semicircular canal. From here it passes posteriorly and laterally along the medial wall of the middle ear. Mastoid or vertical segment extends from the second genu to stylomastoid foramen deep to tympano-mastoid suture line from where the nerve exits out of the temporal bone. During our endoscopic dissection we encountered a grossly anomalous course of facial nerve in which after turning at second genu, the nerve curves posteriorly and lies in the floor of mastoid cavity and traverse’s its whole length of mastoid and instead of moving out of foramen it travels towards sinus plate and then takes another (3rd) turn to travel anteriorly towards the tip of mastoid from where it finally exits.
EN
Objectives: The aim was to evaluate the position of the cochlear implant electrode inside the cochlea and damage to cochlear structures associated with the implantation itself using cone beam computed tomography technique (CBCT). Material and methods: Nine human cadaver temporal bones were used, five were implanted with round window approach and in other four anterior cochleostomy was used for insertion. After implantation the temporal boneswere scanned with CBCT scanner and the images were then analyzed. The degree of insertion damage was evaluated in two-tier scale, where the first degree included damage to basilar membrane, and the second degree covered damage associated with pushing up into the scala vestibuli. Results: The first degree of cochlear damage was noted in three temporal bones implanted with the round window approach and in two with cochleostomy, and the second degree of damage was noted in two and one temporal bones respectively. The analysis did not show any correlations between depth of insertion and degree and extend of damage in both analyzed groups, also no significant differences were found between the two groups. Conclusions: The good quality of the images presents CBCT as a good method for the evaluation of the cochlear implant electrode position in the inner ear structures. CBCT holds the promise for intraoperative imagining during cochlear implantation.
EN
Introduction: Proliferative-inflammatory pathologies may occupy the temporal bone, resulting in: hearing loss, vestibular dysfunction, and neuropathies from cranial nerve compression. Although their occurrence is episodic, the appropriate diagnostic procedure is extremely important to achieve expected therapeutic effect. Aim: The aim of study was characterization of selected proliferative-inflammatory pathologies that may occupy the temporal bone: fibrous dysplasia, inflammatory pseudotumor, osteoradionecrosis, and presentation of diagnostic methods for the differentiation of these diseases as well as discussion on appropriate therapeutic options. Fibrous dysplasia (FD) is a slowly progressive, benign bone disorder of unknown etiology characterized by abnormal proliferation of fibrous tissue. Inflammatory pseudotumor (IPT) is a rare, non-malignant inflammatory process of unknown etiology, characterized by connective tissue proliferation and infiltration of inflammatory cells. Osteoradionecrosis of the temporal bone (TB-ORN) is a rare but potentially fatal complication of radiotherapy for head and neck cancer. Results: Due to the similarity of symptoms with typical inflammatory conditions of middle ear (pain, otorrhea, hearing loss), selected disorders may be a dilemma regarding the diagnosis and proper further treatment. The clinical examination is mandatory, however radiological imaging may demonstrate the presence of specific changes and direct the diagnosis. The computed tomography (CT) of fibrous dysplasia shows the abnormal organization of the bone structure. Magnetic resonance (MRI), as the most sensitive for inflammatory pseudotumors, visualizes inflammatory infiltration in soft tissues. The CT of temporal bone identifies the erosion in the course of osteoradionecrosis. However in all cases the final diagnosis may be establish using histopathological examination and after exclusion of the neoplastic process.
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