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Robust projective outer synchronization of coupled uncertain fractional-order complex networks

100%
Wang J., Zhang Y.
Open Physics
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2013
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vol. 11
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issue 6
813-823
EN
In this work, we propose a novel projective outer synchronization (POS) between unidirectionally coupled uncertain fractional-order complex networks through scalar transmitted signals. Based on the state observer theory, a control law is designed and some criteria are given in terms of linear matrix inequalities which guarantee global robust POS between such networks. Interestingly, in the POS regime, we show that different choices of scaling factor give rise to different outer synchrony, with various special cases including complete outer synchrony, anti-outer synchrony and even a state of amplitude death. Furthermore, it is demonstrated that although stability of POS is irrelevant to the inner-coupling strength, it will affect the convergence speed of POS. In particular, stronger inner synchronization can induce faster POS. The effectiveness of our method is revealed by numerical simulations on fractional-order complex networks with small-world communication topology.
2
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Activity and kinetics studies of yeast alcohol dehydrogenase in a reverse micelle formulated from functional surfactants

61%
Zhang Y., Huang X., Ji G., Li Y., Liu W., Qu Y.
Open Chemistry
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2009
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vol. 7
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issue 4
787-793
EN
Yeast alcohol dehydrogenase (YADH) showed substantial decrease in its catalytic activity due to the strong electrostatic interaction between the head groups of sodium bis(2-ethylhexyl) sulfosuccinate (AOT) and YADH in AOT reverse micelles. However, the catalytic activity of YADH in a nonionic reverse micellar interface (GGDE/TX-100) obtained from a functional nonionic surfactant N-gluconyl glutamic acid didecyl ester (GGDE) and Triton X-100 (TX-100) was higher than that in AOT reverse micelle under the respective optimum conditions. A comparison of the kinetic parameters showed that the turnover number kcat in GGDE/TX-100 reverse micelle was 1.4 times as large as that in AOT reverse micelle, but the Michaelis constants in AOT reverse micelle for ethanol K mB was twice and for coenzyme NAD+ K mA was 5 times higher than their counterparts in GGDE/TX-100 reverse micelle. For the conversion of ethanol, the smaller K mB and larger kcat in GGDE/TX-100 reverse micelle resulted in higher catalytic efficiency kcat/K mB. The stability of YADH in GGDE/TX-100 reverse micelle was also found to be better than that in AOT reverse micelle. They were mainly attributed to the absence of electric charge on the head groups of GGDE and TX-100 in the GGDE/TX-100 reverse micelle. [...]
3
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Upregulation of GRP78 and caspase-12 in diastolic failing heart

61%
Sun Y., Liu G., Song T., Liu F., Kang W., Zhang Y., Ge Z.
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2008
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vol. 55
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issue 3
511-516
EN
Background: The endoplasmic reticulum (ER) fulfills multiple cellular functions. Various stimuli can potentially cause ER stress (ERS). ERS is one of the intrinsic apoptosis pathways and apoptosis plays a critical role in hypertension. Glucose regulated protein 78 (GRP78) has been widely used as a marker for ERS and caspase-12 mediated apoptosis was a specific apoptotic pathway of ER. The expression of GRP78 and caspase-12 remains poorly understood in the diastolic heart failure resulting from hypertension. Methods: We used spontaneously hypertensive rats (SHRs) to establish a model of diastolic heart failure, and performed immunohistochemistry, western blot, and real-time PCR to analyze GRP78 and caspase-12. Results: We found that GRP78 and caspase-12 had enhanced expression at protein and mRNA levels. Conclusions: These results suggest that GRP78 and caspase-12 were upregulated in cardiomyocytes and ERS can contribute to cardiac myocyte apoptosis in the diastolic heart failure resulting from hypertension.
4
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Myocardial remodeling in rats with metabolic syndrome: role of Rho-kinase mediated insulin resistance

52%
Li C.-B., Li X.-X., Chen Y.-G., Gao H.-Q., Bao C.-M., Liu X.-Q., Bu P.-L., Zhang J., Zhang Y., Ji X.-P.
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2012
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vol. 59
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issue 2
249-254
EN
Insulin resistance (IR) plays a critical role in metabolic syndrome (MS). Previous studies have demonstrated that activated ROCK is increased in MS patients. However, the effect of Rho-kinase (ROCK) on IR has not been definitely determined. Thus, the aims of the present study were to determine whether ROCK activation induces IR or affects myocardial structure and function, as well as the possible mechanisms underlying this process. Wistar rats fed high fat, high glucose and high salt diet sewed as model of MS and we used transmission electron microscopy, echocardiogram technology, and terminal deoxynucleotidyl transferase-mediated DNA nick-end labeling staining to identify any myocardial damage. The protein levels of MYPT-1 (characteristic of ROCK activation), IRS-1 and AKT were analyzed by immunohistochemistry and Western blotting. In hearts from MS rats, we found increased protein levels of phospho-MYPT-1 and phospho-IRS-1 (Ser307) and decreased phospho-AKT compared to levels in normal rats. In conclusion, the results suggest that ROCK-mediated IR is involved in the development of myocardial impairments in MS rats and that this effect is mediated probably via the IRS-1/PI3-kinase/AKT pathway.
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