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1
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AC Microcalorimetry of Superconducting MgCNi_3 Single Crystals

100%
Kačmarčík J., Pribulová Z., Samuely P., Marcenat C., Klein T., Jang D., Lee H., Lee H., Lee S.
Acta Physica Polonica A
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2008
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vol. 113
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issue 1
363-366
EN
The low-temperature specific heat of single-crystal samples of superconducting MgCNi_3 with typical dimensions 200 μm were measured for the first time. A computer controlled ac microcalorimeter using an optical fiber and an infrared light-emitting diode as the heat source was used down to 2 K at magnetic fields up to 8 T. The specific heat data suggest a moderate coupling in MgCNi_3.
2
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Point-Contact Spectroscopy of Superconducting MgCNi_3 Single Crystals

100%
Pribulová Z., Szabó P., Kačmarčík J., Samuely P., Jang D., Lee H., Lee H., Lee S.
Acta Physica Polonica A
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2008
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vol. 113
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issue 1
215-218
EN
The first point-contact spectroscopy measurements on MgCNi_3 single crystals of very good quality are presented. The measurements have been performed in the temperature range from 1.5 K up to 10 K and at magnetic fields up to 6 T. The superconducting energy gap Δ of the system is found to be scattered between 1 and 1.2 meV which gives the 2Δ/k T_c ratio between BCS weak-coupling value of ~3.5 and a strong coupling of about 4.2. The excess current evaluated in magnetic field indicates the presence of a single energy gap.
3
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Sources and mechanisms of cytoplasmic oxidative damage in Alzheimer's disease

99%
Marlatt M., Lee H., Perry G., Smith M. A., Zhu X.
Acta Neurobiologiae Experimentalis
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2004
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vol. 64
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issue 1
81-87
EN
While evidence supports a pathogenic and proximal role for oxidative stress in Alzheimer's disease, the causes and consequences of reactive oxygen species that promote oxidative damage have not been directly demonstrated. Co-incident with the reduced energy metabolism during the development of the disease, some of the key mitochondrial enzymes have shown deficient activity in AD neurons, which may lead to increased ROS production. However, we found that oxidative damage occurs primarily within the cytoplasm rather than in mitochondria. Given that SOD activity is increased in AD mitochondria and that metal ions such as iron and copper are enriched in susceptible neurons, we hypothesize that mitochondria, as a source, provide hydrogen peroxide, which, as an intermediate, once in the cytoplasm, will be converted into highly reactive hydroxyl radicals through Fenton reaction in the presence of metal ion and cause damage in cytoplasm.
4
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Preparation Of Fine Grained SiC At Reduced Temperature By Two-Step Sintering

99%
Kim K.-W., Oh K.-S., Lee H., Kim B.-S., Chung T.-J.
Archives of Metallurgy and Materials
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2015
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issue 2
5
Content available remote

The role of metabotropic glutamate receptors in Alzheimer's disease

72%
Lee H., Zhu X., O'Neill M. J., Webber K., Casadesus G., Marlatt M., Raina A. K., Perry G., Smith M. A.
Acta Neurobiologiae Experimentalis
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2004
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vol. 64
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issue 1
89-98
EN
While glutamatergic transmission is severely altered by early degeneration of cortico-cortical connections and hippocampal projections in Alzheimer's disease (AD), the role of glutamate receptors in the pathogenesis of AD is not yet defined clearly. Nonetheless, as reviewed here, the topographical distribution of different types of receptors likely contributes to the regional selective nature of neuronal degeneration. In particular, metabotropic glutamate receptors (mGluR) may contribute the pathogenesis of many neurological conditions and also regulate neuronal vulnerability against cytotoxic stress. Thus, we here discuss the possible role of mGluR in the pathogenesis of AD based on the results from other neurodegenerative diseases that may give us clues to solve the mysterious selective neurodegeneration evident in AD.
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