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DOES GERSTMANN SYNDROME EXIST?

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EN
The aim of the study is to present Gerstmann syndrome, manifested as a neuropsychological deficit resulting from the damage to the parietal lobe of the left hemisphere. Here it is discussed based on the studies conducted mainly since the 1950’s when it attracted considerable interest, as well as and controversy at the same time. The classic symptoms are briefly described, including the clinical tasks useful in any the diagnosis for during the neuropsychological assessment. The paper also presents recent studies and a alternative different proposal for the understanding of this clinical syndrome. Josef Gerstmann described a clinical tetrad in his patients, which was later to be known as Gerstmann syndrome. The symptoms included finger agnosia, agraphia, acalculia and left-right disorientation. He associated the above symptoms with damage to the left angular gyrus, hence the alternative a different name for of the syndrome i.e., the angular gyrus syndrome. The existence of the syndrome was questioned for some time, something which was never approved by Gerstmann. Currently, the occurrence of the syndrome is confirmed by studies. However, the full and pure tetrad of the classic symptoms as observed is not common. The clinical picture of the syndrome often usually remains incomplete and is related to other neuropsychological deficits such as aphasia, which frequently occurs. In modern considerations, the language deficiencies of semantic aphasia are not treated as non- Gerstmann syndrome, disturbing its pure form, but are considered to be a part of Gerstmann syndrome as such.
EN
INTRODUCTION: Carpal tunnel syndrome (CTS) is an upper limb neuropathy that occurs as a result of compression of the median nerve in the carpal tunnel and is the most common mononeuropathy in the general population. The aim of the study was to assess the electrophysiological parameters of the median nerve before and 6 months after surgical treatment of CTS in patients with a history of smoking and comorbidities. MATERIAL AND METHODS: 84 patients with CTS who were eligible for surgery were enrolled in this prospective study. Electrophysiological tests were performed in the patients before and 6 months after surgery for CTS. RESULTS: The results of the study prove that smoking and diabetes significantly worsen the electrophysiological parameters in patients undergoing surgical treatment of CTS. CONCLUSIONS: Smoking and diabetes cause a significantly worse prognosis in patients after surgery for CTS.
PL
WSTĘP: Zespół cieśni nadgarstka (ZCN) jest neuropatią kończyny górnej, pojawiającą się w wyniku ucisku nerwu pośrodkowego w kanale nadgarstka. Celem badania była ocena parametrów elektrofizjologicznych nerwu pośrodkowego przed leczeniem i 6 miesięcy po leczeniu operacyjnym ZCN u pacjentów obciążonych nikotynizmem oraz chorobami współistniejącymi. MATERIAŁ I METODY: Do prospektywnego badania włączono 84 pacjentów z rozpoznanym klinicznie i elektrofizjologicznie ZCN kwalifikowanych do leczenia operacyjnego. U każdego pacjenta przed leczeniem oraz 6 miesięcy po leczeniu operacyjnym ZCN przeprowadzono badanie elektrofizjologiczne. WYNIKI: Uzyskane wyniki dowodzą, że nikotynizm i cukrzyca istotnie pogarszają parametry elektrofizjologicz-ne u pacjentów poddanych leczeniu operacyjnemu ZCN. WNIOSKI: Nikotynizm i cukrzyca są niekorzystnymi rokowniczo czynnikami u osób operowanych z powodu ZCN.
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