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EN
The paper presents configuration interaction (CI) calculations of both the length and velocity forms of the oscillator strengths for 1s2s^{1}S^{e} → 1s2p^{1}P^{0} transition in the He atom. For the first time the agreement between the length and velocity values to within about 0.045% has been achieved.
EN
We study the Hong-Mandel 2n th-order squeezing of the Hermitian operator, X_θ ≡ X₁cosθ + X₂sinθ and amplitude n th-power squeezing of the Hermitian operator, Y_θ^{(n)} ≡ Y₁^{(n)}cosθ + Y₂^{(n)}sinθ in superposed state |ψ⟩ = K[|α,+⟩ + re^{iφ}|0⟩], of vacuum state and even coherent state defined by |α,+⟩ = K_+[|α⟩+|-α⟩]. Here operators X_{1,2} are defined by X₁ + iX₂ = a, operators Y_{1,2}^{(n)} are defined by Y₁^{(n)} + iY₂^{(n)} = aⁿ, a is the annihilation operator, α, θ, r and φ are arbitrary and the only restriction on these is the normalization condition of the superposed state. We show that the Hong-Mandel 2n th-order squeezing and amplitude odd-power squeezing exhibited by even coherent state enhance in its superposition with vacuum state. Variations of these higher-orders squeezing with different parameters near its maxima have also been discussed.
EN
We examined the expression of protein kinase C isoforms in infarcted tissue, penumbra and contralateral brain tissue from 10 patients who died between 1-52 days after ischaemic stroke. Ten patients aged 61-89 years were used in the study. Tissue samples were assayed for protein kinase C activity using a non-radioactive method, and specific isoforms expression determined by Western blotting and staining with anti-PKC polyclonal antibodies. There was a 2-24 fold increase in PKC gamma in the ischaemic penumbra of nine out of 10 patients compared to contralateral tissue. In infarcted tissue expression of PKC gamma was not significantly changed in any of 10 samples but the beta I isoform increased in eight and the beta II in nine patients. There was no significant change in expression in PKC alpha or in infarct or penumbra. Differences in total PKC activity were not specific in seven out of eight patients and it is difficult to estimate their significance. In conclusion after ischaemia there was an altered expression of PKC isoforms with an increase of PKC gammain the surviving penumbra and beta I and beta II in the infarcted core.
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