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2014 | 9 | 2 | 217-222

Article title

Sudden infant death syndrome and perinatal risk factors


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1 - 4 - 2014
13 - 2 - 2014


  • High Nursing Study, Technical College in Bjelovar, Bjelovar, Croatia
  • High Nursing Study, Technical College in Bjelovar, Bjelovar, Croatia
  • High Nursing Study, Technical College in Bjelovar, Bjelovar, Croatia
  • High Nursing Study, Technical College in Bjelovar, Bjelovar, Croatia
  • High Nursing Study, Technical College in Bjelovar, Bjelovar, Croatia


  • [1] Willinger M, James LS, Catz C. Defining the sudden infant death syndrome (SIDS): deliberations of an expert panel convened by the National Institute of Child Health and Human Development. Pediatr Pathol 1991;11:677–684 http://dx.doi.org/10.3109/15513819109065465[Crossref]
  • [2] Corwin MJ. Sudden infant death syndrome: risk factors and risk reduction strategies. UpTo Date, Jun 12, 2012
  • [3] Habek D, Habek JC, Jugović D, Salihagić A. Intrauterine hypoxia and sudden infant death syndrome. Acta Med Croatica 2002;56:109–118 [PubMed]
  • [4] Thach BT. The role of respiratory control disorders in SIDS. Respir Physiol Neurobiol 2005;149:343–353 http://dx.doi.org/10.1016/j.resp.2005.06.011[Crossref]
  • [5] Habek D, Živko M, Selthofer, Kulaš T. Sudden infant death syndrome and perinatal risk. J Perinat Med 2005;33(Suppl 1):261
  • [6] Hunt CE. Gene-environment interactions: implications for sudden unexpected deaths in infancy. Arch Dis Child 2005;90:48–53 http://dx.doi.org/10.1136/adc.2004.051458[Crossref]
  • [7] Guntheroth WG, Spiers PS. The triple risk hypotheses in sudden infant death syndrome. Pediatrics 2002;110:64 http://dx.doi.org/10.1542/peds.110.5.e64[Crossref]
  • [8] Scott CB, Nickerson BG, Sargent CW, Dennies PC, Platzker AC, Keens TG. Diaphragm strength in near-miss sudden infant death syndrome. Pediatrics 1982;69:782–784
  • [9] Highet AR, Berry AM, Bettelheim KA, Goldwater PN. The frequency of molecular detection of virulence genes encoding cytolysin A, high-pathogenicity island and cytolethal distending toxin of Escherichia coli in cases of sudden infant death syndrome does not differ from that in other infant deaths and healthy infants. J Med Microbiol 2009;58:285–289 http://dx.doi.org/10.1099/jmm.0.005322-0[Crossref]
  • [10] Supinski GS, Callahan LA. Hemin prevents cardiac and diaphragm mitochondrial dysfunction in sepsis. Free Radic Biol Med 2006;40:127–137 http://dx.doi.org/10.1016/j.freeradbiomed.2005.09.025[Crossref]
  • [11] Remmers JE, deGroot WJ, Sauerland EK, Anch AM. Pathogenesis of upper airway occlusion during sleep. J Appl Physiol 1978;44:931–938
  • [12] Kinney HC, Feliano JJ. Brain stem research in sudden infant death syndrome. Pediatrician 1988;15:240–243 [PubMed]
  • [13] Del Bigio MR, Becker LE. Microglial aggregation in the dentate gyrus: a marker of mild hypoxic-ischemic brain insult in human infants. Neuropathol Appl Neurobiol 1994;20:144–151 http://dx.doi.org/10.1111/j.1365-2990.1994.tb01173.x[Crossref]
  • [14] Storm H, Nylander G, Saugstad OD. The amount of brainstem gliosis in sudden infant death syndrome victims correlates with maternal cigarette smoking during pregnancy. Acta Paed 1999;1:13–18
  • [15] Valdes Dapnea M. The sudden infant death syndrome: pathologic findings. Clin Perinatol 1992;19:757–772
  • [16] Goldwater PN. Sudden infant death syndrome: a critical review of approaches to research, Arch Dis Child 2003;88:1095–1100 http://dx.doi.org/10.1136/adc.88.12.1095[Crossref]
  • [17] Jones RE. Dysfunctional development of the diaphragm in SIDS and the prone sleeping position. Clin Pediatr 1996;35:173–174 http://dx.doi.org/10.1177/000992289603500317[Crossref]
  • [18] Rehan VK, Nakashima JM, Gutman A, Rubin LP, McCool FD. Effects of the supine and prone position on diaphragm thickness in healthy term infants. Arch Dis Child 2000;83:234–238 http://dx.doi.org/10.1136/adc.83.3.234[Crossref]
  • [19] Paluszynska DA, Harris KA, Thach BT. Influence of sleep position experience on ability of prone-sleeping infants to escape from asphyxiating microenvironments by changing head position. Pediatrics 2004;114:1634–1639 http://dx.doi.org/10.1542/peds.2004-0754[Crossref]
  • [20] Kinney HC, Thach BT. The sudden infant death syndrome. N Engl J Med 2009;361:795–805 http://dx.doi.org/10.1056/NEJMra0803836[Crossref]
  • [21] Valdés-Dapena M, Gilbert-Barness E. Cardiovascular causes for sudden infant death. Padiatr Pathol Mol Med 2002;21:195–211 http://dx.doi.org/10.1080/pdp.[Crossref]
  • [22] Tester DJ, Ackerman MJ. Sudden infant death syndrome: how significant are the cardiac channelopathies? Cardiovasc Res 2005;67:388–396 http://dx.doi.org/10.1016/j.cardiores.2005.02.013[Crossref]
  • [23] Tester DJ, Dura M, Carturan E, et al. A mechanism for sudden infant death syndrome (SIDS): stress induced leak via ryanodine receptors. Heart Rhythmol 2007;4:733–739 http://dx.doi.org/10.1016/j.hrthm.2007.02.026[Crossref]
  • [24] Schwartz PJ, Stramba-Badiale M, Segantini A, et al. Prolongation of the QT interval and the sudden infant death syndrome. N Engl J Med 1998;338:1709–1714 http://dx.doi.org/10.1056/NEJM199806113382401[Crossref]
  • [25] Haas JE, Taylor JA, Bergman AB, et al. Relationship between epidemiologic risk factors and clinicopathologic findings in SIDS. Pediatrics 1993;91:106–112 [PubMed]
  • [26] Mathews, TJ, Menacker, F, MacDorman, MF. Infant mortality statistics from the 2002 period: linked birth/infant death data set. Natl Vital Stat Rep 2004;53:1 [PubMed]

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